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UID:20240426T232233EDT-7327f2Nabo@132.216.177.157
DTSTAMP:20240427T032233Z
DESCRIPTION:Fructose is a simple sugar found in fruit and honey\, but it is
  also used as sweeteners via added sugars\, syrups\, or high fructose corn
  syrup in processed foods or beverages. This is a concern because excessiv
 e fructose intake is linked to obesity and its comorbid diseases like diab
 etes and cardiovascular disease. We show that mice consuming a high fructo
 se diet eat more and gain more weight and body fat than mice consuming cho
 w or a high glucose diet. As the brain is a vital organ mediating the acti
 ons of fructose\, we determined the neuronal maladaptations underlying fru
 ctose-mediated obesity. Interestingly\, fructose feeding increased excitat
 ory drive to hypothalamic neurons known to stimulate food intake\, and thi
 s excitatory drive is comparable to that seen during hunger. This presenta
 tion will examine the synaptic plasticity and whether it may be reversed w
 ith the cessation of fructose feeding. Our findings indicate that central 
 maladaptations following excessive fructose consumption may contribute to 
 overeating and the development of diet-induced obesity.\n\nThe F.C. MacInt
 osh Lectureship supports special seminars in honour of Dr. F.C. MacIntosh.
 \n
DTSTART:20200214T160000Z
DTEND:20200214T170000Z
LOCATION:Room 1034\, McIntyre Medical Building\, CA\, QC\, Montreal\, H3G 1
 Y6\, 3655 promenade Sir William Osler
SUMMARY:Seminar - Fructose and the Hungry Brain
URL:https://www.mcgill.ca/physiology/channels/event/seminar-fructose-and-hu
 ngry-brain-302286
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